How to cure atherosclerosis?

Atherosclerosis is largely related to mineral and protein metabolism, and this connection is explained by the physiology of the vascular wall and lipid metabolism.

Protein Factor

Blood vessels consist not only of endothelium and lipids but also of protein structures — collagen and elastin. In the case of protein deficiency, the synthesis of these structures decreases, making the vessels brittle and permeable. In response to endothelial damage, the body "patches" them with lipids (primarily low-density lipoproteins), which initiates the atherosclerotic process.

Moreover, low levels of albumin in plasma disrupt the transport of minerals (calcium, magnesium, iron), exacerbating their imbalance.

Calcium Metabolism

Atherosclerosis is accompanied by pathological calcification of blood vessels. Here, the important factor is not the quantity of calcium but its proper distribution:

• With a deficiency of vitamin K2, calcium is not retained in the bones and "leaves" for the vascular wall, where it deposits in plaques.
• A lack of magnesium worsens the situation: magnesium is a natural antagonist of calcium, relaxing blood vessels and preventing calcium deposition in soft tissues.
• Disruption of stomach acidity (hypoacidity) hinders the absorption of calcium in ionic form and further disturbs the balance.

Iron and Its Role

Iron metabolism also directly affects:

• An excess of iron accelerates lipid peroxidation (LPO), damaging the endothelium through free radicals.
• A deficiency of iron reduces the synthesis of antioxidant defense enzymes (e.g., catalase), which also increases oxidative stress.
• Thus, both excess and deficiency of iron can enhance the progression of atherosclerosis.

General Pathogenesis Scheme:

1. Endothelial damage (stress, toxins, protein deficiency).
2. Disruption of calcium and iron transport → destabilization of the vascular wall.
3. The body "repairs" the vessel with lipids, gradually forming a plaque.
4. If protein and magnesium deficiency persists, calcium becomes fixed in the plaque, making it hard and dangerous.

Correction:

• Sufficient intake of animal protein (collagen amino acids, albumins).
• Control of calcium + magnesium + vitamin K2 + vitamin D3 for proper calcium distribution.
• Maintenance of normal levels of iron and ferritin, avoiding both overload and deficiency.
• Support of the antioxidant system (selenium, zinc, vitamins C and E) to reduce lipid oxidation.